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Elife:出生时的并发症与大脑持久的化学变化有关

2017-11-30 海北 MedSci原创

围产期脑损伤(包括海马损伤)会引起啮齿类动物多巴胺功能的持久变化,但是如果这种情况发生在人类身上,我们还不知道会导致怎么样的后果。

围产期脑损伤(包括海马损伤)会引起啮齿类动物多巴胺功能的持久变化,但是如果这种情况发生在人类身上,我们还不知道会导致什么样的后果。

来自英国国王学院的研究人员对非常早产出生并且有围产期脑损伤的成年人,与非围产期脑损伤的非常早产的成年人,足月出生的年龄相匹配的对照组进行了比较。研究人员使用[18F] -DOPA PET和结构MRI对其进行了探究。

研究人员发现,与非围产期脑损伤组(Cohen's d = 1.36p = 0.02)以及足月对照组(Cohen's d = 1.07p = 0.01)相比,围产期脑损伤组的多巴胺合成能力降低。和足月对照组相比,围产期脑损伤组的海马体积也有所减少(Cohen's d = 1.17p = 0.01)。海马体积和纹状体多巴胺合成能力正相关(r = 0.344p = 0.03)。

该研究是将人类新生儿海马损伤与成年多巴胺功能障碍联系起来的第一个证据,并提供了将早期生活危险因素与成年精神疾病联系起来的潜在机制。


原始出处:

Sean Froudist-Walsh et al. The effect of perinatal brain injury on dopaminergic function and hippocampal volume in adult lifeeLife, 2017; 6 DOI: 10.7554/eLife.29088


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    2017-12-23 clmlylxy
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    2018-06-26 膀胱癌
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    2018-05-30 丁鹏鹏
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    2017-11-30 1ddf0692m34(暂无匿称)

    学习了.涨知识

    0

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    2017-11-30 神功盖世

    学习

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    2017-11-30 sunfeifeiyang

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