Bis(ethylmaltolato)oxidovanadium (IV) mitigates neuronal apoptosis resulted from amyloid-beta induced endoplasmic reticulum stress through activating peroxisome proliferator-activated receptor gamma-MedSci.cn

Bis(ethylmaltolato)oxidovanadium (IV) mitigates neuronal apoptosis resulted from amyloid-beta induced endoplasmic reticulum stress through activating peroxisome proliferator-activated receptor gamma

He, ZJ; Wang, MH; Zhao, QH; Li, XQ; Liu, PG; Ren, BY; Wu, C; Du, XB; Li, N; Liu, Q

Li, N (corresponding author), Shenzhen Univ, Coll Life Sci & Oceanog, Shenzhen Bay Lab, Shenzhen 518055, Peoples R China.; Liu, Q (corresponding author), Shenzhen Univ, Shenzhen Hong Kong Inst Brain Sci, Coll Life Sci & Oceanog, Shenzhen Fundamental Res Inst, Shenzhen, Peoples R China.

Abstract

Neuronal apoptosis caused by amyloid-beta (A beta) overproduction is one of the most important pathological features in Alzheimer's disease (AD). Endo......

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Bis(ethylmaltolato)oxidovanadium (IV) mitigates neuronal apoptosis resulted from amyloid-beta induced endoplasmic reticulum stress through activating peroxisome proliferator-activated receptor gamma

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