盘点：2018年4月Gastroenterology研究精选

2018-04-02 zhangfan MedSci原创

zhangfan

西宁市第一人民医院医生/ 住院医师

2018年4月Gastroenterology研究精选

【1】封面文章--非恶性肠道息肉手术率增加

DOI： https：//doi.org/10.1053/j.gastro.2018.01.003

近日研究人员就美国非恶性肠息肉手术的变化趋势进行了考察。

研究收集并分析了2000-14年间相关病人样本资料。研究期间，总计进行了1230458例肠道息肉及肿瘤手术，其中25%是非恶性息肉手术。非恶性息肉手术率由2000年的每100000患者5.9例上升至2014年的每100000患者9.4例，而结肠癌手术率则显著下降，由每100000患者31.5例下降至24.7例，20-79岁人群、男性、女性以及所有种族中，非恶性肠道息肉手术率均显著增加。

【2】糖尿病患者服用阿司匹林或可降低胰腺癌风险

DOI： https：//doi.org/10.1053/j.gastro.2017.12.001

近日研究人员考察了阿司匹林以及其他非甾体抗炎药(NSAIDs )与胰腺癌风险之间的关系。

1122名患者在420万随访年中确诊胰腺癌。阿司匹林以及其他NSAIDs不能降低胰腺癌风险。亚组分析显示，仅糖尿病患者规律的服用阿司匹林可降低胰腺癌风险(rr 0.71)。病例对照研究中，确诊前血浆水杨酸浓度与胰腺癌风险不相关(浓度前1/5与后1/5患者OR， 1.08)。

【3】幽门螺杆菌减少胃腺体胆固醇干扰γ-干扰素信号通路和免疫炎症反应

DOI： https：//doi.org/10.1053/j.gastro.2017.12.008

幽门螺杆菌可以在胃粘膜中存在几十年，除了引发炎症反应之外，胃定殖和T细胞活化过程中均需要幽门螺杆菌表达的胆固醇-α-葡萄糖基转移酶（cgt编码）。近日研究人员考察了cgt对胃上皮细胞与宿主免疫应答的影响。

原发性胃癌细胞经野生型幽门螺杆菌感染后，IFNG依赖信号通路阻断。受感染细胞胆固醇耗尽，导致导致JAK和STAT1通路磷酸化活化减少。信号通路被IFNB、IL6以及 IL22阻断，导致调节T细胞功能的细胞因子和抗微生物肽的基因表达降低。这一生理机制允许幽门螺杆菌在感染细胞附近持续存在，同时仅在邻近的、非感染的上皮中诱导炎症。PMSS1感染后胃组织IFNG水平升高，但干扰素反应基因表达水平未显著上升。PMSS1Δcgt 小鼠的IFNG响应基因IRF1的表达水平高于PMSS1小鼠。Ifngr1-/-小鼠PMSS1的定植率显著提高。

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#插入话题

插入图片

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2019-01-02 jktdtl

#Gastroenterol#

62 0
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2018-05-09 许安

#GAS#

63 0
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2018-09-05 zhouqu_8

#AST#

58 0
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2018-04-04 chaojitidian

#Gastroenterology#

53 0
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2018-04-03 易水河

阿司匹林的用途很多.这么多年过去了仍然可以挖掘出新的治疗作用

84 0
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2018-04-03 秀红

学习了

82 0

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